- This is a rather interesting view point on the current and past 1918 flu pandemic. Lawrence Broxmeyer MD writes :
The CDC has a short memory. There was much the same “Influenza” talk when in 1990, a new multi-drug-resistant (MDR) tuberculosis outbreak took place in a large Miami municipal hospital. Soon thereafter, similar outbreaks in three New York City hospitals left many sufferers dying within weeks. By 1992, approximately two years later, drug-resistant tuberculosis had spread to seventeen US states, with mini-epidemics in Florida, Michigan, New York, California, Texas, Massachusetts, and Pennsylvania and was reported, by the international media, as out of control. Viral forms of swine, avian and human TB can be transmitted from one species to another.
“There have been about three influenza pandemics in each century for the last 300 years”, points out Lawrence Broxmeyer MD . “Yet when the horrendous H1N1 1918 pandemic first got national attention in America in April, 1918, authorities didn’t even considered Influenza or “the Flu” lethal enough to be a reportable disease.”
Yet ironically, this very 1918 “Influenza” strain itself held clinical and epidemiologic similarities to the influenza pandemics of 1889, 1847, and even earlier. So it can be easily understood why some experts, from 1918 to the present, validly question how such an explosively fatal disease could have been or is presently caused by “Influenza”.
Even Lancet, one of the most prominent medical journals in the world, calls the Mexican epidemic “flu-like” because frankly we do not have enough facts or verification in to call it anything else.
No one can deny the similarities between the onset of the 1918 epidemic and that of today. Yet a Press Release, issued on August 19, 2008, by the National Institute of Allergy and Infectious Diseases (NIAID), contains a striking finding and conclusion: The 20 to 40 million deaths worldwide from the great 1918 Influenza (“Flu”) Pandemic were NOT due to “flu” or a virus, but to pneumonia caused by massive bacterial infection.
Subsequently, a study published in JAMA by Talbot and Moore in 2000 showed that Mexican immigrants to the US have the highest case rates for tuberculosis among foreign born persons.
The research of Lawrence Broxmeyer MD, first proclaimed that the 1918 pandemic was due to bacteria, particularly mutant forms of flu-like fowl, swine, bovine, and human tuberculosis (TB) bacteria. These forms of tuberculosis are often viral-like, mutate frequently and can “skip” from one species to another. Moreover the antibodies from such viral TB forms react in the compliment fixation and later “viral” assays. They also grow on cultures which are supposed to grow only viruses.
In 2006 Medline paper published in Elsevier’s Medical Hypothesis, Lawrence Broxmeyer, MD wrote: “Influenza is Italian for “influence”, Latin: influentia. It used to be thought that the disease was caused by a bad influence from the heavens. Influenza was called a virus long, long before it was proven to be one.” Elsevier is a major medical publisher which also publishes The Lancet.
In 2005, an article in the New England Journal of Medicine estimated that a recurrence of the 1918 influenza epidemic could kill between 180 million and 360 million people worldwide. It is yet to be seen whether the present pandemic will be what that journal had in mind. Yet similar to the present Swine episode, a large part of the previous bird-flu hysteria was fostered by a distrust among the lay and scientific community regarding the actual state of our knowledge regarding the bird flu or H5N1 and the killer “Influenza” Pandemic of 1918 that it was compared to. Such distrust was not completely unfounded. Traditionally, “flu” does not kill. Experts, including Peter Palese of the Mount Sinai School of Medicine in Manhattan, remind us that even in 1992, millions in China already had antibodies to the Bird Flu H5N1, meaning that they had contracted it and that their immune system had little trouble fending it off.
In 2000 Dr. Andrew Noymer and Michel Garenne, UC Berkeley demographers, reported convincing statistics showing that undetected tuberculosis may have been the real killer in the 1918 flu epidemic. Although scientists have recently spun that a “new” strain of “Influenza” strikes healthy young people, flu traditionally attacks the old and the infirm. The 1918 killer, on the other hand, went after men and women in their prime, between 20-40, prime ages for a TB onslaught. Flu didn’t traditionally show the male preference recorded in 1918. TB did. With TB routinely, there was extremely low mortality between the ages of 5 and 15, also reflected in 1918. Besides, Noymer pointed out, Influenza typically came in the winter, this one began in the late spring and summer. The first autopsy of a 1918 pandemic victim occurred in Chicago in April, the very same month that the Mexican swine “flu” came to our attention .This mystery 1918 disease would subside, but come back in the fall with a vengeance.
Yet, Gorgas, Head of the US Army Medical Corp. at that time, insisted on referencing it as ”Influenza”, an old and heretofore not that deadly disease. Furthermore, argued Noymer, flu traditionally gave mild to moderate fever, but in the 1918 pandemic, patients could spike to 105 or 106 degrees, not uncharacteristic for an acute attack of tuberculosis.
And so, Noymer and Garenne, aware of recent attempts to isolate the “Influenza virus” on human cadavers and their specimens, nevertheless concluded that: “Frustratingly, these findings have not answered the question why the 1918 virus was so virulent, nor do they offer an explanation for the unusual age profile of deaths”.
“By 1918″, Lawrence Broxmeyer, MD continued, “it could be said, in so far as tuberculosis was concerned, that the world was a supersaturated sponge ready to ignite and that among its most vulnerable parts was the very Midwest where the 1918 unknown pandemic began. A critical mass, much like the Mexican experience, had been reached. It is theorized that the lethal pig epidemic that began in 1918 Kansas just prior to the first human outbreak was a disease of avian and human tuberculosis genetically combined through mycobacteriophage interchange, with the pig susceptible to both, and as its involuntary living culture medium.” This to is much like the inception of the Mexican epidemic.
In commenting on the NIAID report, Lawrence Broxmeyer MD writes that euphemisms like “pneumonia”, “bronchitis” , “chest ailment”, “the flu” etc. have been with us since time immemorial to describe tubercular involvement. This was noted in 1944 in pathologist Arnold Rich’s classic test, “The Pathogenesis of Tuberculosis.” It is also well known that bacterial secondary infection in TB cases is common. And since NIAD’s 2008 report there can be little doubt that bacterial infection was the cutting edge of death in 1918.
Whatever the “connection” between TB bacteria , influenza, and a virus-caused “flu” epidemic, the August, 2008 report by the NIAID is a reminder that epidemics and pandemics are complex disease processes, which should not simply be assumed to be due merely to a “virus” of great ferocity.
- Quantitative Easing is gathering steam. More money out of thin air. Inflation is definitely in the near horizon. It won’t be long before we have a global monetary collapse. Countries of the world will adopt a beggar thy neighbour policy of competitive devaluations. Reuters reports :
The Bank of England stepped up its campaign to boost the struggling economy, raising the size of its asset purchase programme on Thursday in a surprise move tantamount to printing an extra 50 billion pounds to get banks lending again.
The central bank kept rates at a record low of 0.5 percent as expected, but surprised markets by moving swiftly to reassure them that its scheme to buy up government and corporate bonds with newly-created money would last at least 3 months more.
Traders had been worried the Bank would wait until next week’s quarterly Inflation Report, or even next month’s policy meeting, before deciding whether to expand the initial 75 billion pounds of asset purchases, which looked set for completion around the end of this month.
“We are a little taken aback by the decision to increase the quantitative easing target by 50 billion pounds. We had thought it more likely the Monetary Policy Committee would sit and wait to assess the impact of the existing programme rather than expand it right away,” said Philip Shaw, economist at Investec.
“Clearly, committee members have been spooked by some poor backward-looking domestic and international economic data. But the statement also points out that forward looking numbers are showing promising signs.”
ASSET PURCHASE BOOST
The Bank of England’s quantitative easing policy aims to help the economy by ensuring inflation stays close to its 2 percent target even when output looks likely to suffer its worst year since the aftermath of World War Two.
So far, the Bank has spent about 50 billion pounds of the 75 billion pounds it said in March it would spend on government and corporate debt. Chancellor Alistair Darling has authorised the Bank to spend up to 150 billion.
Buying debt with newly created money boosts the money supply and in theory allows banks to lend more freely. It is intended to limit the undershoot in inflation — which fell to 2.9 percent in March — the Bank expects later this year.
“That stimulus should in due course lead to a recovery in economic growth, bringing inflation back towards the 2 percent target. But the timing and strength of that recovery is highly uncertain,” the central bank said. The Bank added it would continue to keep its quantitative easing policy under review.
- ECB looks like following the path of QE. Bloomberg reports :
European Central Bank President Jean- Claude Trichet said the ECB unanimously agreed on a 60 billion- euro ($80.5 billion) plan to buy bonds as officials step up their response to the worst recession since World War II.
“The Governing Council has decided in principle that the eurosystem will purchase euro-denominated covered bonds issued in the euro area,” Trichet told reporters in Frankfurt. He said the bank’s main interest rate, which it cut by a quarter point to a record low of 1 percent today, is appropriate and that the ECB will extend its unlimited auctions of funds to banks.
ECB officials have spent the past months bickering over whether to fight a recession by purchasing assets, with Bundesbank President Axel Weber leading resistance to such a move. The U.S. Federal Reserve, the Bank of England and Bank of Japan have lowered rates close to zero and are already buying bonds, effectively printing money to reflate their economies in a policy known as quantitative easing.
- YouTube :
As the first cases of the 2009 Mexican flu outbreak were being reported, Dr. Leonard Horowitz’s telephone began to ring requesting his comments. A few hours later, after investigating vaccine makers involved, the Harvard-trained emerging diseases expert uploaded to YouTube a 10 minute SPECIAL REPORT that will go down in history as the classic scam buster. Within a few hours of alerting his network, thousands of people viewed this riveting video, more than 90,000 in 30 hours, topping Google’s search list throughout the three-day fright.
Dr. Horowitz, the worlds leading governmental whistle-blower in consumer health advocacy, exposed the agents and agencies most implicated in creating and releasing the extraordinary virus and publicity needed to place the world at high alert. They claimed this was a rapidly spreading combination of swine flu, avian flu, Spanish flu, and more. Dr. Horowitz caught them with incriminating scientific publications, cited their names, published their references, and called for an immediately international investigation into this global genocide. Among those exposed were Americas disease-busters at the CDC and an allied private company, NOVAVAX, maker of the Johnny-on-the-spot vaccine. Needless to say, the inside players initially made, and then lost, millions in the stock market.